GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F

177 Lu-DOTATATE selectively killed hSSTr2 + teratomas as demonstrated

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

Essential oil yield estimation by Gas chromatography–mass spectrometry (GC– MS) after Methyl jasmonate (MeJA) elicitation in in vitro cultivated tissues of Coriandrum sativum L.

2018-Metabolomics Digital 6, PDF, Metabolomics

Identification and quantitation of unsaturated fatty acid isomers using the ZenoTOF 7600 system

EU Regulation Compliant GC-MS/MS Method Package for Dioxins in Foods - Features : Shimadzu Scientific Instruments

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

Determination of enantiomeric and stable isotope ratio fingerprints of active secondary metabolites in neroli (Citrus aurantium L.) essential oils for authentication by multidimensional gas chromatography and GC-C/P-IRMS - ScienceDirect

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

Frontiers Histone methyltransferase SETD2: An epigenetic driver in clear cell renal cell carcinoma

New-Generation, High-Precision Isotope Ratio Mass Spectrometry System Delivers Analysis for Geosciences, Nuclear Safeguards, and Medical Research Applications

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F cells. (A)

Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect